We’re excited to announce our latest peer-reviewed publication now available in the open-access American Journal of Physiology-Cell Physiology (Impact Factor: 5.5). Our paper, “Activation of ankrd1a expression marks newly forming myofibers and regulates muscle cell differentiation in adult zebrafish skeletal muscle repair”, is officially published and accessible online: DOI: https://doi.org/10.1152/ajpcell.00807.2024
Publication Highlights
This new study from our ZEBARR research team uncovers the function of the ankrd1a gene in controlling skeletal muscle repair. Key findings include:
- ankrd1a activation marks newly forming muscle fibers
- ankrd1a acts as a “brake”—preventing new muscle cells that repopulate the wound from differentiating and maturing too quickly, which could otherwise disrupt proper healing.
Why It Matters
Uncovering the mechanisms by which ankrd1a regulates skeletal muscle repair could contribute to developing therapies for muscle injuries, degenerative muscle diseases, or age-related muscle loss.
Availability of the Study
The complete study represents the results of extensive work by our research team and is now available to the scientific community.
As part of our commitment to sharing scientific knowledge, this paper has been published in open-access format, meaning it is freely available to all researchers and interested readers worldwide. We invite you to read our study and learn more about this important discovery.
